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By D. Springer

ISBN-10: 9535100203

ISBN-13: 9789535100201

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1995). , 1995). , 1996). , 1996). In addiction, as reviewed by Zoeller et al. , 2002), many TDs can disrupt TH signaling without affecting circulating levels of THs. , 2002). , 1998). However, the developmental effects of TD exposure in experimental animals are not fully consistent with mechanism attributable to hypothyroidism. , 1995) similarly to that observed in hypothyroid rats. , 1998). , 1995; Hood & Klaassen, 2000). , 1995). These and other observations suggest that different TDs or their mixtures may produce heterogeneous disrupting effects on the thyroid system also without affecting circulating T4/T3 levels.

Furthermore, chemicals are not currently tested specifically for their ability to mimic, disrupt, or otherwise act as hormone agonists or antagonists, except on research basis. Finally, more studies are crucial to fill in the research gaps regarding permanent endocrine and neurological outcome in next generations exposed to background TDs. 6. S. & Morita, M. (2005). Structural requirements for the interaction of 91 hydroxylated polychlorinated biphenyls with estrogen and thyroid hormone receptors.

T. (2005). Maternal thyroid hormone increases HES expression in the fetal rat brain: an effect mimicked by exposure to a 34 A New Look at Hypothyroidism mixture of polychlorinated biphenyls (PCBs). Brain Research. Developmental Brain Research, 156, 1, (April 2005), pp. 13-22. A. D. (1994). Reduction of thyroid hormone levels and alteration of thyroid function by four representative UDP-glucuronosyltransferase inducers in rats. Toxicology and Applied Pharmacology, 128, 1, (September 1994), pp. 9-17.

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A New Look at Hypothyroidism by D. Springer


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